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Friday, November 20, 2020 | History

3 edition of Glucoregulation and work performance in gluconeogenesis-inhibited iron deficient rats found in the catalog.

Glucoregulation and work performance in gluconeogenesis-inhibited iron deficient rats

Glucoregulation and work performance in gluconeogenesis-inhibited iron deficient rats

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  • 2 Currently reading

Published .
Written in English

    Subjects:
  • Energy metabolism.,
  • Gluconeogenesis.,
  • Lactic acid.,
  • Iron in the body.,
  • Rats.

  • Edition Notes

    Statementby Jon Karl Linderman.
    The Physical Object
    FormatMicroform
    Paginationxii, 147 leaves
    Number of Pages147
    ID Numbers
    Open LibraryOL16908710M


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Glucoregulation and work performance in gluconeogenesis-inhibited iron deficient rats Download PDF EPUB FB2

Get this from a library. Glucoregulation and work performance in gluconeogenesis-inhibited iron deficient rats. [Jon Karl Linderman]. iron deficiency and work performance Download iron deficiency and work performance or read online books in PDF, EPUB, Tuebl, and Mobi Format.

Click Download or Read Online button to get iron deficiency and work performance book now. This site is like a library, Use search box in the widget to get ebook that you want. Maintenance of euglycemia is impaired in gluconeogenesis-inhibited iron-deficient rats at rest and during exercise.

Linderman JK(1), Brooks GA, Rodriguez RE, Dallman PR. Author information: (1)Department of Human Biodynamics, University of California, Berkeley Cited by: 4. Glucoregulation and work performance in gluconeogenesis-inhibited iron deficient rats by Jon Karl Linderman.

QP PH MFICHE Influence of post-exercise glucose ingestion on plasma potassium levels and ECG measurements submitted by Hedy Reynolds.

Reduced running endurance in gluconeogenesis-inhibited rats. John-Alder HB, McAllister RM, Terjung RL. The functional significance of gluconeogenesis in prolonging endurance during submaximal activity was assessed in untrained and endurance-trained rats.

Gluconeogenesis was inhibited at the phosphoenolpyruvate carboxykinase reaction by 3 Cited by:   Serum lactate in rats was elevated 56% by 2,5-anhydro-D-mannitol, but this could be prevented by dichloroacetate ( mg/kg) or thiamin ( mg/kg). In hepatocytes from fasted rats, 1 mM 2,5-anhydro-D-mannitol inhibited gluconeogenesis from a mixture of alanine, lactate, and pyruvate.

It makes sense that AMP would inhibit the process, as gluconeogenesis requires ATP for it to work. Glucagon stimulates the gluconeogenesis process in order for the liver to "share" this excess energy with the rest of the body. When the book says "fasting state", it is talking about the needs of muscles and other organs that are not the liver.

- [Instructor] At its most simplistic level, regulation of metabolic pathways inside of the body is really just a fancy word for a balancing act that's occurring in the body. So, to illustrate this, I have a seesaw and we've been learning about two metabolic pathways: glycolysis, which is the process of breaking down glucose into pyruvate; and.

a hormone that is secreted by the adrenal gland in response to physical or mental stress, as from fear, and is regulated by the autonomic nervous system; its release causes an increase in heart rate, blood pressure, and respiratory rate, as well as raising glucose levels in the blood for use as fuel when more alertness or greater physical effort is needed; also called adrenaline.

chemical. Gluconeogenesis (GNG) is a metabolic pathway that results in the generation of glucose from certain non-carbohydrate carbon substrates.

From breakdown of proteins, these substrates include glucogenic amino acids (although not ketogenic amino acids); from breakdown of lipids (such as triglycerides), they include glycerol, odd-chain fatty acids (although not even-chain fatty acids, see below.